Id: | acc0201 |
Group: | 2sens |
Protein: | JAK2 |
Gene Symbol: | JAK2 |
Protein Id: | O60674 |
Protein Name: | JAK2_HUMAN |
PTM: | phosphorylation |
Site: | Tyr1007 |
Site Sequence: | TKVLPQDKEYYKVKEPGESPI |
Disease Category: | Cancer |
Disease: | Prostate Cancer |
Disease Subtype: | |
Disease Cellline: | DU145 |
Disease Info: | |
Drug: | EC-70124 |
Drug Info: | "Epirubicin is an anthracycline chemotherapeutic agent that inhibits cancer cell growth by interfering with DNA replication and cell division, primarily used in the treatment of breast cancer, ovarian cancer, and gastric cancer. Cyclophosphamide is an alkylating agent that suppresses DNA and RNA synthesis, exhibiting both antineoplastic and immunomodulatory effects, often combined with epirubicin in EC chemotherapy regimens to enhance efficacy and reduce drug resistance. " |
Effect: | modulate |
Effect Info: | "EC - 70124 inhibits the phosphorylation of IκB, STAT3 (Tyr705) and JAK2 (Tyr1007/1008), thereby suppressing tumors." |
Note: | |
Score: | 4.0 |
Pubmed(PMID): | 26826115 |
Sentence Index: | 26826115_5-6 |
Sentence: | "In this study, we show that EC-70124 blocked concomitantly NF-kappaB and STAT3 in prostate cancer cells and particularly prostate CSCs, which exhibited overactivation of these transcription factors. Phosphorylation of IkB and STAT3 (Tyr705), the immediate targets of IKKbeta and JAK2, respectively, was rapidly inhibited in vitro by EC-70124 at concentrations that were well below plasma levels in mice." |
Sequence & Structure:
MGMACLTMTEMEGTSTSSIYQNGDISGNANSMKQIDPVLQVYLYHSLGKSEADYLTFPSGEYVAEEICIAASKACGITPVYHNMFALMSETERIWYPPNHVFHIDESTRHNVLYRIRFYFPRWYCSGSNRAYRHGISRGAEAPLLDDFVMSYLFAQWRHDFVHGWIKVPVTHETQEECLGMAVLDMMRIAKENDQTPLAIYNSISYKTFLPKCIRAKIQDYHILTRKRIRYRFRRFIQQFSQCKATARNLKLKYLINLETLQSAFYTEKFEVKEPGSGPSGEEIFATIIITGNGGIQWSRGKHKESETLTEQDLQLYCDFPNIIDVSIKQANQEGSNESRVVTIHKQDGKNLEIELSSLREALSFVSLIDGYYRLTADAHHYLCKEVAPPAVLENIQSNCHGPISMDFAISKLKKAGNQTGLYVLRCSPKDFNKYFLTFAVERENVIEYKHCLITKNENEEYNLSGTKKNFSSLKDLLNCYQMETVRSDNIIFQFTKCCPPKPKDKSNLLVFRTNGVSDVPTSPTLQRPTHMNQMVFHKIRNEDLIFNESLGQGTFTKIFKGVRREVGDYGQLHETEVLLKVLDKAHRNYSESFFEAASMMSKLSHKHLVLNYGVCVCGDENILVQEFVKFGSLDTYLKKNKNCINILWKLEVAKQLAWAMHFLEENTLIHGNVCAKNILLIREEDRKTGNPPFIKLSDPGISITVLPKDILQERIPWVPPECIENPKNLNLATDKWSFGTTLWEICSGGDKPLSALDSQRKLQFYEDRHQLPAPKWAELANLINNCMDYEPDFRPSFRAIIRDLNSLFTPDYELLTENDMLPNMRIGALGFSGAFEDRDPTQFEERHLKFLQQLGKGNFGSVEMCRYDPLQDNTGEVVAVKKLQHSTEEHLRDFEREIEILKSLQHDNIVKYKGVCYSAGRRNLKLIMEYLPYGSLRDYLQKHKERIDHIKLLQYTSQICKGMEYLGTKRYIHRDLATRNILVENENRVKIGDFGLTKVLPQDKEYYKVKEPGESPIFWYAPESLTESKFSVASDVWSFGVVLYELFTYIEKSKSPPAEFMRMIGNDKQGQMIVFHLIELLKNNGRLPRPDGCPDEIYMIMTECWNNNVNQRPSFRDLALRVDQIRDNMAG
Select PDB:
Target | Drug name | MOA | Phase | Status | Disease | Source |
---|---|---|---|---|---|---|
JAK2 | BARICITINIB | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | immune system disease | ATC |
JAK2 | FILGOTINIB | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | immune system disease | ATC |
JAK2 | RUXOLITINIB | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | neoplasm | ATC |
JAK2 | PACRITINIB | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | neoplasm | ATC |
JAK2 | FEDRATINIB | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | neoplasm | ATC |
JAK2 | TOFACITINIB CITRATE | Janus Kinase (JAK) inhibitor | 4 | - | rheumatoid arthritis | DailyMed DailyMed |
JAK2 | BARICITINIB | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | rheumatoid arthritis | FDA DailyMed EMA |
JAK2 | TOFACITINIB CITRATE | Janus Kinase (JAK) inhibitor | 4 | Recruiting | rheumatoid arthritis | ClinicalTrials |
JAK2 | BARICITINIB | Tyrosine-protein kinase JAK2 inhibitor | 4 | Recruiting | rheumatoid arthritis | ClinicalTrials ClinicalTrials ClinicalTrials |
JAK2 | BARICITINIB | Tyrosine-protein kinase JAK2 inhibitor | 4 | Terminated | rheumatoid arthritis | ClinicalTrials |
JAK2 | BARICITINIB | Tyrosine-protein kinase JAK2 inhibitor | 4 | Active, not recruiting | rheumatoid arthritis | ClinicalTrials |
JAK2 | BARICITINIB | Tyrosine-protein kinase JAK2 inhibitor | 4 | Completed | rheumatoid arthritis | ClinicalTrials |
JAK2 | FILGOTINIB MALEATE | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | rheumatoid arthritis | EMA |
JAK2 | UPADACITINIB HEMIHYDRATE | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | rheumatoid arthritis | FDA DailyMed |
JAK2 | PACRITINIB CITRATE | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | primary myelofibrosis | FDA |
JAK2 | RUXOLITINIB PHOSPHATE | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | polycythemia vera | EMA DailyMed |
JAK2 | PACRITINIB CITRATE | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | polycythemia vera | FDA |
JAK2 | RUXOLITINIB PHOSPHATE | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | primary myelofibrosis | DailyMed |
JAK2 | FEDRATINIB HYDROCHLORIDE | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | primary myelofibrosis | FDA EMA |
JAK2 | TOFACITINIB CITRATE | Janus Kinase (JAK) inhibitor | 4 | - | psoriatic arthritis | DailyMed |
JAK2 | RUXOLITINIB PHOSPHATE | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | myeloproliferative disorder | EMA |
JAK2 | FEDRATINIB HYDROCHLORIDE | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | myeloproliferative disorder | EMA |
JAK2 | RUXOLITINIB | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | Eczematoid dermatitis | ATC |
JAK2 | FEDRATINIB HYDROCHLORIDE | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | Myelofibrosis | DailyMed |
JAK2 | RUXOLITINIB PHOSPHATE | Tyrosine-protein kinase JAK2 inhibitor | 4 | - | Myelofibrosis | DailyMed |
Note: Only show clinically investigational or approved drugs with protein targets.
Protein Tractability:
source: Open TargetsPTM Intensity:
source: CPTACJAK2-Tyr570 | |
---|---|
Cancer | Intensity |
BRCA | |
COAD | |
HGSC | |
ccRCC | |
GBM | -0.417 |
HNSC | -0.724 |
LUAD | |
LUSC | 1.141 |
non_ccRCC | |
PDAC | |
UCEC |
PTM-Disease Association:
source: PTMDResidue | Position | State | Disease | Class | PMID |
---|---|---|---|---|---|
Y | 1007 | U | Prostate cancer/carcinoma/adenocarcinoma | Phosphorylation | 22307624 |
State Note: Based on the distinct PTM states in diseases, PTMD classified all disease-associated PTMs into six classes, including whether the up-regulation (U) or down-regulation (D) of PTM levels, the absence (A) or presence (P) of PTMs, and the creation (C) or disruption (N) of PTM sites are associated with diseases.
PTM-Drug Perturbation Response:
source: DecryptMNo data.
Function score:
source: funscoRNo data.