Id: | acc2483 |
Group: | 2sens |
Protein: | VEGFR2 |
Gene Symbol: | KDR |
Protein Id: | P35968 |
Protein Name: | VGFR2_HUMAN |
PTM: | phosphorylation |
Site: | Tyr1059 |
Site Sequence: | ARDIYKDPDYVRKGDARLPLK |
Disease Category: | Cancer |
Disease: | Hepatocellular Carcinoma |
Disease Subtype: | |
Disease Cellline: | SMMC-7721 |
Disease Info: | |
Drug: | I131-metuximab |
Drug Info: | "Iodine (???I) Metuximab Injection is a radioactive iodine-labeled monoclonal antibody produced by HAb18 hybridoma cells, used for the treatment of unresectable or recurrent primary hepatocellular carcinoma and advanced hepatocellular carcinoma patients who are unsuitable for or have failed transarterial chemoembolization (TACE) therapy. " |
Effect: | modulate |
Effect Info: | I131-metuximab (I131-mab) enhances its anti-cancer activity by inhibiting VEGFR-2 phosphorylation and reversing the epithelial-mesenchymal transition of hepatocellular carcinoma (HCC) cells. |
Note: | site unclear |
Score: | 4.0 |
Pubmed(PMID): | 29210217 |
Sentence Index: | 29210217_5-6 |
Sentence: | "Mechanically, this inhibition effect was mainly mediated by the antibody component part of I131 -mab, which could reverse the epithelial-mesenchymal transition of HCC cells partially by suppressing the phosphorylation of VEGFR-2. The inhibitory effect of I131 on HCC cell proliferation and invasion is limited, whereas, when combined with metuximab, I131 significantly enhanced the sensitivity of HCC cells to CD147-mab and consequently reinforced the anticancer effects of CD147-mab, suggesting that the two components of I131 -mab exerted synergistic anti-HCC capability." |
Sequence & Structure:
MQSKVLLAVALWLCVETRAASVGLPSVSLDLPRLSIQKDILTIKANTTLQITCRGQRDLDWLWPNNQSGSEQRVEVTECSDGLFCKTLTIPKVIGNDTGAYKCFYRETDLASVIYVYVQDYRSPFIASVSDQHGVVYITENKNKTVVIPCLGSISNLNVSLCARYPEKRFVPDGNRISWDSKKGFTIPSYMISYAGMVFCEAKINDESYQSIMYIVVVVGYRIYDVVLSPSHGIELSVGEKLVLNCTARTELNVGIDFNWEYPSSKHQHKKLVNRDLKTQSGSEMKKFLSTLTIDGVTRSDQGLYTCAASSGLMTKKNSTFVRVHEKPFVAFGSGMESLVEATVGERVRIPAKYLGYPPPEIKWYKNGIPLESNHTIKAGHVLTIMEVSERDTGNYTVILTNPISKEKQSHVVSLVVYVPPQIGEKSLISPVDSYQYGTTQTLTCTVYAIPPPHHIHWYWQLEEECANEPSQAVSVTNPYPCEEWRSVEDFQGGNKIEVNKNQFALIEGKNKTVSTLVIQAANVSALYKCEAVNKVGRGERVISFHVTRGPEITLQPDMQPTEQESVSLWCTADRSTFENLTWYKLGPQPLPIHVGELPTPVCKNLDTLWKLNATMFSNSTNDILIMELKNASLQDQGDYVCLAQDRKTKKRHCVVRQLTVLERVAPTITGNLENQTTSIGESIEVSCTASGNPPPQIMWFKDNETLVEDSGIVLKDGNRNLTIRRVRKEDEGLYTCQACSVLGCAKVEAFFIIEGAQEKTNLEIIILVGTAVIAMFFWLLLVIILRTVKRANGGELKTGYLSIVMDPDELPLDEHCERLPYDASKWEFPRDRLKLGKPLGRGAFGQVIEADAFGIDKTATCRTVAVKMLKEGATHSEHRALMSELKILIHIGHHLNVVNLLGACTKPGGPLMVIVEFCKFGNLSTYLRSKRNEFVPYKTKGARFRQGKDYVGAIPVDLKRRLDSITSSQSSASSGFVEEKSLSDVEEEEAPEDLYKDFLTLEHLICYSFQVAKGMEFLASRKCIHRDLAARNILLSEKNVVKICDFGLARDIYKDPDYVRKGDARLPLKWMAPETIFDRVYTIQSDVWSFGVLLWEIFSLGASPYPGVKIDEEFCRRLKEGTRMRAPDYTTPEMYQTMLDCWHGEPSQRPTFSELVEHLGNLLQANAQQDGKDYIVLPISETLSMEEDSGLSLPTSPVSCMEEEEVCDPKFHYDNTAGISQYLQNSKRKSRPVSVKTFEDIPLEEPEVKVIPDDNQTDSGMVLASEELKTLEDRTKLSPSFGGMVPSKSRESVASEGSNQTSGYQSGYHSDDTDTTVYSSEEAELLKLIEIGVQTGSTAQILQPDSGTTLSSPPV
Select PDB:
Target | Drug name | MOA | Phase | Status | Disease | Source |
---|---|---|---|---|---|---|
KDR | CABOZANTINIB S-MALATE | Vascular endothelial growth factor receptor 2 inhibitor | 4 | - | hepatocellular carcinoma | EMA |
KDR | MIDOSTAURIN | Vascular endothelial growth factor receptor 2 inhibitor | 4 | - | acute myeloid leukemia | FDA |
KDR | RAMUCIRUMAB | Vascular endothelial growth factor receptor 2 inhibitor | 4 | - | neoplasm | ATC |
KDR | LENVATINIB | Vascular endothelial growth factor receptor inhibitor | 4 | - | neoplasm | ATC |
KDR | AXITINIB | Vascular endothelial growth factor receptor inhibitor | 4 | - | neoplasm | ATC |
KDR | MIDOSTAURIN | Vascular endothelial growth factor receptor 2 inhibitor | 4 | - | neoplasm | ATC |
KDR | REGORAFENIB | Vascular endothelial growth factor receptor inhibitor | 4 | - | neoplasm | ATC |
KDR | CABOZANTINIB | Vascular endothelial growth factor receptor 2 inhibitor | 4 | - | neoplasm | ATC |
KDR | VANDETANIB | Vascular endothelial growth factor receptor inhibitor | 4 | - | neoplasm | ATC |
KDR | CABOZANTINIB S-MALATE | Vascular endothelial growth factor receptor 2 inhibitor | 4 | - | renal cell carcinoma | EMA DailyMed |
KDR | LENVATINIB MESYLATE | Vascular endothelial growth factor receptor inhibitor | 4 | - | renal cell carcinoma | EMA DailyMed |
KDR | TIVOZANIB HYDROCHLORIDE | Vascular endothelial growth factor receptor inhibitor | 4 | - | renal cell carcinoma | FDA DailyMed |
KDR | SUNITINIB | Vascular endothelial growth factor receptor inhibitor | 4 | - | neoplasm | ATC |
KDR | SUNITINIB | Vascular endothelial growth factor receptor inhibitor | 4 | Completed | neoplasm | ClinicalTrials |
KDR | SORAFENIB TOSYLATE | Vascular endothelial growth factor receptor inhibitor | 4 | - | renal cell carcinoma | DailyMed |
KDR | PAZOPANIB HYDROCHLORIDE | Vascular endothelial growth factor receptor inhibitor | 4 | - | renal cell carcinoma | FDA DailyMed |
KDR | AXITINIB | Vascular endothelial growth factor receptor inhibitor | 4 | - | renal cell carcinoma | DailyMed DailyMed EMA |
KDR | SUNITINIB MALATE | Vascular endothelial growth factor receptor inhibitor | 4 | - | renal cell carcinoma | DailyMed |
KDR | SUNITINIB MALATE | Vascular endothelial growth factor receptor inhibitor | 4 | Completed | renal cell carcinoma | ClinicalTrials |
KDR | PAZOPANIB HYDROCHLORIDE | Vascular endothelial growth factor receptor inhibitor | 4 | - | sarcoma | FDA DailyMed |
KDR | NINTEDANIB ESYLATE | Vascular endothelial growth factor receptor inhibitor | 4 | - | systemic scleroderma | DailyMed |
KDR | SUNITINIB | Vascular endothelial growth factor receptor inhibitor | 4 | - | renal cell carcinoma | DailyMed EMA |
KDR | SUNITINIB | Vascular endothelial growth factor receptor inhibitor | 4 | Completed | renal cell carcinoma | ClinicalTrials ClinicalTrials |
KDR | SUNITINIB | Vascular endothelial growth factor receptor inhibitor | 4 | Terminated | renal cell carcinoma | ClinicalTrials ClinicalTrials |
KDR | NINTEDANIB ESYLATE | Vascular endothelial growth factor receptor inhibitor | 4 | - | idiopathic pulmonary fibrosis | DailyMed |
Note: Only show clinically investigational or approved drugs with protein targets.
Protein Tractability:
source: Open TargetsPTM Intensity:
source: CPTACNo data.
PTM-Disease Association:
source: PTMDResidue | Position | State | Disease | Class | PMID |
---|---|---|---|---|---|
Y | 951 | D | Head and neck squamous cell carcinoma | Phosphorylation | 21281788 |
Y | 996 | D | Head and neck squamous cell carcinoma | Phosphorylation | 21281788 |
- | - | D | Papillary thyroid cancer | Ubiquitination | 22711876 |
K | 1041 | P | Prostate cancer/carcinoma/adenocarcinoma | Methylation | 24300896 |
K | 1041 | P | Melanoma | Methylation | 24300896 |
- | - | U | Breast cancer/tumor/carcinoma | Phosphorylation | 10371349 |
Y | 1175 | U | Pulmonary hypertension | Phosphorylation | 37024132 |
State Note: Based on the distinct PTM states in diseases, PTMD classified all disease-associated PTMs into six classes, including whether the up-regulation (U) or down-regulation (D) of PTM levels, the absence (A) or presence (P) of PTMs, and the creation (C) or disruption (N) of PTM sites are associated with diseases.
PTM-Drug Perturbation Response:
source: DecryptMNo data.
Function score:
source: funscoRNo data.