Id: | acc3708 |
Group: | 1sens |
Protein: | STAT1 |
Gene Symbol: | Stat1 |
Protein Id: | P42224 |
Protein Name: | STAT1_HUMAN |
PTM: | phosphorylation |
Site: | unclear |
Site Sequence: | |
Disease Category: | Cardiovascular and circulatory system diseases |
Disease: | Myocardial Ischemic Injury |
Disease Subtype: | |
Disease Cellline: | |
Disease Info: | |
Drug: | dehydroascorbic acid (DHA) |
Drug Info: | Dehydroascorbic acid (DHA) is an oxidized form of ascorbic acid. It can play various roles in biological systems. |
Effect: | modulate |
Effect Info: | "DHA reduces cardiomyocyte apoptosis and has a protective effect against myocardial ischemia in rats by inhibiting protein phosphorylation (e.g., phosphorylation of Jak2, STAT1, and STAT5b), increasing the phosphorylation of anti - apoptotic proteins (e.g., Bcl - 2, Bcl - xl), and suppressing the activation of apoptosis - related proteins (e.g., Bax, caspase - 3). " |
Note: | |
Score: | 4.0 |
Pubmed(PMID): | 15454281 |
Sentence Index: | 15454281_8 |
Sentence: | "DHA treatment also inhibited Jak2, STAT1, and STAT5 phosphorylationation, and increased STAT3 phosphorylationation, in hypoxic cardiomyocytes and ischemic myocardial tissue." |
Sequence & Structure:
MSQWYELQQLDSKFLEQVHQLYDDSFPMEIRQYLAQWLEKQDWEHAANDVSFATIRFHDLLSQLDDQYSRFSLENNFLLQHNIRKSKRNLQDNFQEDPIQMSMIIYSCLKEERKILENAQRFNQAQSGNIQSTVMLDKQKELDSKVRNVKDKVMCIEHEIKSLEDLQDEYDFKCKTLQNREHETNGVAKSDQKQEQLLLKKMYLMLDNKRKEVVHKIIELLNVTELTQNALINDELVEWKRRQQSACIGGPPNACLDQLQNWFTIVAESLQQVRQQLKKLEELEQKYTYEHDPITKNKQVLWDRTFSLFQQLIQSSFVVERQPCMPTHPQRPLVLKTGVQFTVKLRLLVKLQELNYNLKVKVLFDKDVNERNTVKGFRKFNILGTHTKVMNMEESTNGSLAAEFRHLQLKEQKNAGTRTNEGPLIVTEELHSLSFETQLCQPGLVIDLETTSLPVVVISNVSQLPSGWASILWYNMLVAEPRNLSFFLTPPCARWAQLSEVLSWQFSSVTKRGLNVDQLNMLGEKLLGPNASPDGLIPWTRFCKENINDKNFPFWLWIESILELIKKHLLPLWNDGCIMGFISKERERALLKDQQPGTFLLRFSESSREGAITFTWVERSQNGGEPDFHAVEPYTKKELSAVTFPDIIRNYKVMAAENIPENPLKYLYPNIDKDHAFGKYYSRPKEAPEPMELDGPKGTGYIKTELISVSEVHPSRLQTTDNLLPMSPEEFDEVSRIVGSVEFDSMMNTV
Select PDB:
No data.
Protein Tractability:
source: Open TargetsPTM Intensity:
source: CPTACNo data.
PTM-Disease Association:
source: PTMDResidue | Position | State | Disease | Class | PMID |
---|---|---|---|---|---|
- | - | D | Melanoma | Phosphorylation | 12421984 |
- | - | D | Lymphoma | Phosphorylation | 12421984 |
- | - | D | Esophageal carcinoma | Ubiquitination | 30506884 |
- | - | P | Neuroblastoma | Phosphorylation | 14982729 |
- | - | P | Epidermoid carcinoma | Phosphorylation | 23178573 |
- | - | P | Oral squamous cell carcinoma | Phosphorylation | 23992782 |
- | - | U | Glioma | Acetylation | 34784299 |
- | - | U | Chronic colitis | Phosphorylation | 37211384 |
- | - | U | Chronic lymphocytic leukemia | Phosphorylation | 23936412 |
State Note: Based on the distinct PTM states in diseases, PTMD classified all disease-associated PTMs into six classes, including whether the up-regulation (U) or down-regulation (D) of PTM levels, the absence (A) or presence (P) of PTMs, and the creation (C) or disruption (N) of PTM sites are associated with diseases.
PTM-Drug Perturbation Response:
source: DecryptMNo data.
Function score:
source: funscoRNo data.